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JID for Clinicians: Looking to the future of infectious disease therapeutics

Last Updated

January 23, 2026

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IDSA’s Journal of Infectious Diseases has launched a monthly roundup of JID papers with direct relevance to clinicians. Read on to learn more about advances in treating Shiga toxin-producing E. coli infections, the effectiveness of high-dose influenza vaccination and other research ready to inform clinical practice. (Titles and summaries are adapted from the January 2026 issue of JID.)

Virulent Bacteriophages for Controlling Shiga Toxin–Producing Escherichia coli Without Inducing Toxin Production

Companion editorial: Virulent Bacteriophages for Controlling Shiga Toxin–Producing Escherichia coli (STEC) Without Inducing Toxin Production

Shiga toxin-producing E. coli infections cause serious, life-threatening consequences, especially in children, that can be enhanced by antibiotic therapy. This study provides evidence that treatment of STEC infections with virulent, lytic bacteriophages may yield a new therapeutic to safely mitigate STEC O157:H7 infections.

Relative Effectiveness of High-Dose Versus Standard-Dose Influenza Vaccination Against Hospitalizations and Deaths According to Frailty Score: A Post Hoc Analysis of the DANFLU-1 Randomized Trial

High dose (vs. standard dose) influenza vaccination reduced first and recurrent hospitalizations for pneumonia or influenza irrespective of frailty scores in this post-hoc analysis of a randomized feasibility trial (2021-2022 influenza season) in 12,477 older adults aged 65-79 years. High-dose influenza vaccination reduced all-cause mortality in individuals with low frailty scores. Collectively, high-dose influenza vaccination may be preferred for older individuals.

Insights on Recurrent and Sequential Clostridioides difficile Infections From Genomic Surveillance in Minnesota, USA, 2019–2021

In the largest C. difficile infection genomic dataset to date, ~65% of recurrent CDI is associated with the same relapsing C. difficile strain, including ~50% of patients with recurrent CID beyond the eight-week limit of the current case definition for recurrent CDI. In sequential C. difficile strain analyses, gain or loss of putative mobile genetic elements occurs in ~46% of genome pairs. This study confirms that the majority of recurrent CDI infections are due to relapse of the initial, sometimes evolving C. difficile strain or, in some, reinfection with a new C. difficile strain.

 

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